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Here’s a quick overview of the pros and cons of mixing doxycycline and alcohol. Learn about the dangers and potential consequences and discover why Doxycycline is the only antibiotic with this unique factor. Doxycycline does share some common side effects with alcohol and we have additional at: Visit https://www.addict-help.com/doxycycline-and-alcohol/

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A study found that most people believe that mixing antibiotics with alcohol will either stop the medication working or make you sick. But what really happens?

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Here is a step-by-step video on how to register your company with FMCSA Clearinghouse.

I go over the clearinghouse registration portal and the FMCS portal. You have to log into your FMCSA Web Portal first and authorize yourself as the Clearinghouse contact rep.

It’s just a few extra steps but I go over the entire process here.

FMCSA Clearinghouse registration: https://clearinghouse.fmcsa.dot.gov/
FMCSA web portal: https://portal.fmcsa.dot.gov/login
_____________
For New MC#!
You will have a new entrant audit within the first 12 months of operating. Mandated by FMCSA.

You can learn more on our website and download our free checklist to be prepared.
👉USDOT New Entrant Audit Checklist:
✅https://www.truckersauthority.com/new-entrant-audit-checklist

Need a Trucking Insurance Quote For Your Authority?
Give our friends at Gold River Brokerage a call!
Speak to a trained USDOT, Licensed Transporation Insurance professional.
👉Call For A Quote Today: (702) 660-9769
👉https://goldriverinsurance.com/trucking/quotes/

******************************
Looking for an ELD company?
Check out Motive ELD,
Highly Recommended- Discount Link – https://mbsy.co/3qJ7GL (we do get a referral commission)
They offer more than just ELD, check them out: https://mbsy.co/3qJ7GL

_______________________________________________________
USDOT & MC Number is not the only part of the process.

These are the most common permits/steps you need to complete your trucking permits.

✅Business Setup (Corporation or Sole Proprietor)
✅USDOT Number
✅Motor Carrier Number
✅BOC-3
✅UCR
✅Drug and Alcohol Consortium Program
✅State Permit
✅Apportioned Registration
✅IFTA Registration

Additional Permits:
✅SCAC Code
✅KYHUT
✅Oregon Weight Permit
✅NM Weight Permit
✅KYU Weight Permit
✅CA Number
✅MCP
✅TXDMV Number

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Sleep paralysis can be a scary experience, but this video is here to help clear some of the fear and confusion surrounding the strange phenomenon. The video explores what sleep paralysis really is, its biological cause, and also presents some tips to help prevent sleep paralysis.

This video was created by McMaster students Minahil Jabeen, Dhruvika Joshi, Prabhjot Khabra and Vanessa Lombardo in collaboration with the McMaster Demystifying Medicine Program.

Subscribe to the McMaster Demystifying Medicine YouTube channel: https://www.youtube.com/c/DemystifyingMedicine

This video is provided for general and educational information only. Please consult your health care provider for Information about your health.

Copyright McMaster University 2017

References:

Denis, D., French, C., & Gregory, A. (2017). A systematic review of variables associated with sleep paralysis. Sleep Medicine Reviews. http://dx.doi.org/10.1016/j.smrv.2017.05.005

Denis, D., French, C. C., Rowe, R., Zavos, H., Nolan, P. M., Parsons, M. J., & Gregory, A. M. (2015). A twin and molecular genetics study of sleep paralysis and associated factors. Journal of sleep research, 24(4), 438-446.

Fukuda, K. (2005). Emotions during sleep paralysis and dreaming. Sleep And Biological Rhythms, 3(3), 166-168. http://dx.doi.org/10.1111/j.1479-8425.2005.00172.x

Golzari, S., Khodadoust, K., Alakbarli, F., Ghabili, K., Islambulchilar, Z., & Shoja, M. et al. (2012). Sleep paralysis in medieval Persia – the Hidayat of Akhawayni (?-983 AD). Neuropsychiatric Disease and Treatment, 229-234. http://dx.doi.org/10.2147/ndt.s28231

Guilleminault C., and Fromherz S.: Narcolepsy: diagnosis and management. In Kryger M.H., Roth T., and Dement W.C. (eds): Principles and practice of sleep medicine, 4th edition. Philadelphia (PA): Elsevier Saunders, 2005. pp. 780-790

Kompanje, E. (2008). ‘The devil lay upon her and held her down’Hypnagogic hallucinations and sleep paralysis described by the Dutch physician Isbrand van Diemerbroeck (1609-1674) in 1664. Journal of Sleep Research, 17(4), 464-467. http://dx.doi.org/10.1111/j.1365-2869.2008.00672.x

Lu, J., Sherman, D., Devor, M., & Saper, C. (2006). A putative flip–flop switch for control of REM sleep. Nature, 441(7093), 589-594. http://dx.doi.org/10.1038/nature04767

National Institutes of Health. (2017). What is REM sleep?. Eunice Kennedy Shriver National Institute of Child Health and Human Development. Retrieved 7 November 2017, from https://www.nichd.nih.gov/health/topics/sleep/conditioninfo/Pages/rem-sleep.aspx#top

Ohayon, M., Priest, R., Caulet, M., & Guilleminault, C. (1996). Hypnagogic and hypnopompic hallucinations: pathological phenomena?. The British Journal Of Psychiatry, 169(4), 459-467. http://dx.doi.org/10.1192/bjp.169.4.459

Ohayon M.M., Zulley J., Guilleminault C., et al: Prevalence and pathologic associations of sleep paralysis in the general population. Neurology 1999; 52: pp. 1194-1200

Plante, D. T., & Winkelman, J. W. (2008). Parasomnias: psychiatric considerations. Sleep Medicine Clinics, 3(2), 217-229.

Sharpless, B., & Barber, J. (2011). Lifetime prevalence rates of sleep paralysis: A systematic review. Sleep Medicine Reviews, 15(5), 311-315. http://dx.doi.org/10.1016/j.smrv.2011.01.007

Sharpless, B. A., & Grom, J. L. (2014). Isolated Sleep Paralysis: Fear, Prevention, and Disruption. Behavioral Sleep Medicine, 1–6. doi:10.1080/15402002.2014.963583

Solomonova, E. (2017). Sleep Paralysis: phenomenology, neurophysiology and treatment. In The Oxford Handbook of Spontaneous Thought: Mind-Wandering, Creativity, Dreaming, and Clinical Conditions. Oxford University Press. Editors: Kieran Fox, Kalina Christoff

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(USMLE topics) Alcoholic fatty liver, alcoholic hepatitis, and cirrhosis: Signs and symptoms, risk factors, pathophysiology, complications, diagnosis and treatment.
This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/all-animations/digestive-system-videos/-/medias/012159d6-87f0-425d-bd0d-37b284ff287a-alcoholic-liver-disease-narrated-animation
©Alila Medical Media. All rights reserved.
Voice by : Marty Henne
Support us on Patreon and get early access to videos and free image downloads: patreon.com/AlilaMedicalMedia
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Alcoholic liver disease is liver damage caused by alcohol abuse. The risk of developing liver disease correlates with the amount and duration of alcohol use. Daily drinking poses a higher risk than binge drinking. Other risk factors include gender, genetics, and obesity.
Alcoholic liver disease includes 3 disorders that develop in sequence: alcoholic fatty liver, alcoholic hepatitis, and cirrhosis.
Alcoholic fatty liver is accumulation of liver fat as a result of alcohol use. The liver is usually enlarged but not tender. There are often no symptoms and the condition can be reversed if the patient stops drinking.
Alcoholic hepatitis is liver inflammation. Symptoms can range from mild to severe and may include fever, jaundice, fatigue, and a tender, painful and enlarged liver.
Cirrhosis is when a large amount of hepatic tissue is permanently replaced with non-functional scar tissue, known as fibrosis. Symptoms range from those of alcoholic hepatitis to those of end-stage liver disease. The liver eventually shrinks. Cirrhosis cannot be reversed.
The liver is the major site of alcohol metabolism. Two main pathways are involved: alcohol dehydrogenase, ADH; and cytochrome P-450 2E1, or CYP-2E1.
ADH converts alcohol to acetaldehyde. A second enzyme, acetaldehyde dehydrogenase, ALDH, then metabolizes acetaldehyde to acetate. People who have low levels of ALDH are more susceptible to toxic effects of acetaldehyde. Both of these reactions convert NAD+ to NADH. An increased NADH/NAD+ ratio promotes fatty liver by inhibiting fatty acid oxidation and stimulating fatty acid synthesis.
In addition, acetaldehyde also promotes production of enzymes involved in fatty acid synthesis, and inhibits expression of enzymes implicated in fat oxidation. Alcohol use has also been shown to reduce export of fat from the liver. All these events lead to fat accumulation in hepatic cells.
Chronic alcohol use upregulates the CYP-2E1 pathway. This pathway generates harmful reactive oxygen species, which can damage proteins and DNA. The effect is exaggerated in patients who are deficient in antioxidants due to malnutrition. Chronic alcohol exposure also activates hepatic macrophages, triggering inflammation. In addition, acetaldehyde can bind to cellular proteins, forming so-called adducts that are seen as foreign antigens by the immune system, provoking the body’s inflammatory attacks.
Chronic inflammation and subsequent attempts at tissue repair lead to formation of scar tissue. As a result, the liver internal structure is disrupted, impairing its functions. Scarring of liver tissue also obstructs blood flow, causing high blood pressure in the portal vein that brings blood from the intestine to the liver.
As liver functions decline, toxins that are normally removed by the liver can now reach the general circulation and pass into the brain, producing symptoms such as confusion, drowsiness, tremor, or even coma, in a condition known as hepatic encephalopathy. Portal hypertension may cause variceal bleeding, enlarged spleen, and abdominal distension.
Diagnosis is usually based on signs of liver dysfunction, other symptoms related to alcohol use, history of heavy drinking, liver function tests and complete blood count.
Abstinence is the best treatment. Supportive care includes good nutrition and vitamin supplements. Corticosteroids may be used to reduce inflammation but their effectiveness is till debatable. Liver transplantation may be considered for abstinent patients with severe liver damage.
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Sleep Apnea Signs & Symptoms (& Why They Occur) | Central, Obstructive & Mixed Sleep Apnea

Sleep apnea is a term for several conditions involving decreased airflow during sleep. There are 3 main types of sleep apnea including central sleep apnea (CSA), obstructive sleep apnea (OSA), and mixed sleep apnea (MSA), which all cause very similar signs and symptoms, although with some differences. In this lesson, we discuss the signs and symptoms of sleep apnea and why those signs and symptoms occur.

I hope you find this lesson helpful. If you do, please like and subscribe for more lessons like this one!

JJ

**MEDICAL LEGAL DISCLAIMER**: JJ Medicine does not provide medical advice, and the information available on this channel does not offer a diagnosis or advice regarding treatment. Information presented in these lessons is for educational purposes ONLY, and information presented here is not to be used as an alternative to a healthcare professional’s diagnosis and treatment of any person/animal. Only a physician or other licensed healthcare professional are able to determine the requirement for medical assistance to be given to a patient. Please seek the advice of your physician or other licensed healthcare provider if you have any questions regarding a medical condition.

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*Check Out Some of My Other Lessons*

Medical Terminology – The Basics – Lesson 1:

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Fatty Acid Synthesis Pathway:

Wnt/B Catenin Signaling Pathway:

Upper vs. Lower Motor Neuron Lesions:

Lesson on the Purine Synthesis and Salvage Pathway:

Gastrulation | Formation of Germ Layers:

Introductory lesson on Autophagy (Macroautophagy):

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